Prior therapies have focused on effects, not on causes. They have tried to help symptoms or wished to remove amyloid or tau proteins.
Michael Fossel MD, PhD Founder, President
Research shows that cell aging triggers the gradual accumulation of amyloid, as well as the loss of neurons that defines Alzheimer's.
As our cells age, the rate at which we recycle molecules — like beta amyloid — slows down. As recycling slows, there is a gradual accumulation of beta amyloid, resulting in microaggregates, amyloid plaque, and clinical disease.
Telomerase resets the abnormal pattern of gene expression that results in Alzheimer's disease. We can make a difference. Join us.
Telomere shortening modulates gene expression, including the genes that drive Alzheimer's disease.